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Last year, for example, American researchers demonstrated that alpha lipoic acid (ALA) supplementation in older racehorses reduced the oxidative stress burden even under light training loads (4) , while a number of other animal studies have shown that acetyl L-carnitine (ALC)/ALA supplementation reduces oxidative stress and improves mitochondrial function in a number of tissues, including brain, muscle and heart

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Bile acid synthesis is the primary mechanism by which excess cholesterol is eliminated from the body (Shah, 2003)

Long-term supplementations potential link to increased cardiovascular risk through TMAO production deserves attention

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