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In a zebrafish model, compared with C3-carnitine, C18-carnitine and C18:1-carnitine can induce the inhibition of OXPHOS, interfere with the energy supply of cardiomyocytes, and lead to a significant impairment of cardiac systolic function, which manifests as a decreased heart rate and reduced shortening of the atrial and ventricular fractions [21]

J Biol Chem 280(26):2525025257

This effect mainly occurs through the mechanisms that lead to quicker improvement and recovery of brain energy production and, as a result, reduction in lactic acid generation in early postischemic reperfusion [37]

The slope of this control reaction was subtracted to calculate the specific MSA depletion activity in the linear range of the assay

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