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Across cysteamine-, stress-, and NSAID-induced ulcer models, BPC-157 administration consistently promotes healing without requiring acid suppression

Autophagy receptors (P62, NBR1, NDP52, OPTN, TAX1BP1) specifically recognize ubiquitin chains via their LC3-interacting regions, binding to microtubule-associated protein 1A/1B-light chain 3 (LC3) on autophagosomal membranes and directing the engulfment of damaged mitochondria

suggests that TB-500 may interact with cell movement as it binds globular actin (G-actin) and may modulate how actin filaments assemble to plausibly make endothelial cells more able to change shape, migrate, and form multicellular structures

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[DOI] [PMC free article] [PubMed] [Google Scholar] 225.Hausmann M., Obermeier F., Paper D., Balan K., Dunger N., Menzel K., Falk W., Schoelmerich J., Herfarth H., Rogler G