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Experimental interference with CaMKIIGluN2B coupling markedly reduces receptor mobility and attenuates LTP, highlighting a feedback mechanism in which NMDAR activation drives structural synaptic remodeling that supports memory encoding

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Furthermore, polar interactions among residues E200, S204, and N207 of mVP35d, and residues R774, Q325, T795, and K328 of MARL further enhance the structural stability of the NTP entry channel (Fig

2022;222(Pt B):272943

In the absence of crosslinking, all FcRs are incapable of signaling, except for FcIR