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Regardless of their origin, AGE accumulation results in dysfunctional proteins, which are the underlying cause of numerous human diseases 2,3,4,5

Patient-derived xenograft (PDX) cells that survived after treatment with various anticancer drugs showed lower risk scores (RS), lower KRAS expression levels, lower activation states of the RAS-MAPK signaling pathway, and significantly different expressions of ion channel transport genes, which may be related to drug resistance mechanisms (Kim et al., 2015)

Phylogenetic tree construction and conserved motif analysis The GST protein sequences of A

Neurochem Res 48 , 33163326 (2023)