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Extensive evidence has confirmed that specific ferroptosis-related metabolic signals or byproducts were involved in the function, viability, and differentiation of multiple T-cell subgroups, impacting their reactivity to inflammation, infection and tumor immunity ( As was reported recently in tumor immunity, the ferroptotic interaction between CD8+ T cell-secreted IFN- and tumor cells is known to induce the elicitation of tumor ferroptosis and reinforcement of anti-tumor activity ( Besides, regulatory T (Treg) cells with a deletion of PDK1 have been implicated in the generation of excessive ROS, which consequently leads to amplified ferroptosis of themselves (53)

The authors stated that the high levels of GFAP in combination with normal S-100 protein concentrations in CSF indicate reactive astrogliosis in the CNS

[Google Scholar] 41.Zller B., Li X., Sundquist J., Sundquist K

& Hbartner, C

Kalaras et al