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Description
Its copper-binding chemistry has made it a reference compound across regenerative biology, particularly for redox-mediated cellular signalling and matrix-remodelling studies
Furthermore, expression profiling using real-time PCR, western blotting and GSTM2 overexpression following transient knockdown of GSTM1 in HeLa cells confirmed that the absence of GSTM1 activity can be compensated for by the overexpression of GSTM2.
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