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c-Met signaling induces a reprogramming network and supports the glioblastoma stem-like phenotype
Mice lacking apoA-I develop autoimmunity when challenged with a high cholesterol (diet and background, Ldlr -/- ), which includes T cell activation and production of autoantibodies (821,822)
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In particular, systemic administration often results in rapid clearance, low bioavailability, and poor targeting specificity
