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Additionally, BWN heart masses were significantly increased in the PoWeR cohorts compared to the Sed cohorts and were unaffected by NNMTi treatment relative to controls (Supplemental Fig

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The interplay between HAS3-mediated HA synthesis and HYAL1-driven degradation further exemplifies intrinsic temporal control of ECM remodeling, providing a natural biological model that parallels HAGSH scaffold responsiveness [19]