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The important aspect to see while treating tumor patients with MET inhibitor is that tumor derived tumor necrosis factor- (TNF-) or any other inflammatory stimuli enhances the MET activation in human neutrophils and leads to their transmigration across an activated endothelium and releases free radicals, which can inhibit the cancer cell growth [76]

This activation is further associated with the accumulation of extracellular matrix components and proliferation of myofibroblasts, ultimately contributing to the degeneration and necrosis of hepatocytes (70-73)

[PubMed] [Google Scholar] Coloma J., Pitot H.C