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Description
Autophagy and gastrointestinal cancers: the behind the scenes role of long non-coding RNAs in initiation, progression, and treatment resistance
This reliance on correlative evidence from disease contexts has reduced CHAC enzymes to mere stress markers and overlooked their potential as dynamically regulated metabolic integrators
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1 exhibits an expression pattern and functional profile more analogous to mammalian CHAC1, suggesting a role in amino acid metabolism or signaling pathways
effectively improve bioavailability, cellular uptake, and anti-cancer mechanisms of emodin
