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330,331 Therefore, the selective inhibition of PMN-MDSCs by FATP2 presents a novel therapeutic target for cancer treatment
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For example, Quant et.al showed that the attachment of succinyl-CoA to the catalytic cysteine residue on HMGCS2 blocks the binding of acetoacetyl-CoA to the substrate
Aug 6 2021;100(31):e26706
