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A single molecular entity simultaneously activates both the GLP-1 receptor and the amylin receptor (calcitonin receptor complex), combining two complementary satiety and metabolic pathways: GLP-1 receptor activation : Glucose-dependent insulin secretion, glucagon suppression, delayed gastric emptying, and central appetite reduction through hypothalamic signaling Amylin receptor activation : Additional satiety signaling through the area postrema, complementary gastric emptying delay, and glucagon suppression through a distinct mechanism The unimolecular design means both receptor activations occur at a fixed ratio determined by the molecular structure

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As shown in Figure 5a, TGM2 actively catalyzed a reaction in which biotinylated svL4 was linked to polylysine

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Elevated IOP causes lamina cribrosa distortionimpairing neurotrophic transportbut disease progression can occur at normal or effectively treated IOP levels, highlighting other pathogenic mechanisms that may be at play