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In parallel, increasing emphasis on regulated and reproducible synthesis strategies aims to reduce batch-to-batch variability, and improve biological evaluation dependability [91, 103]

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Circadian disruption also coexists with Huntington's disease (HD)

Protective effects of astragaloside IV on uric acid-induced pancreatic -cell injury through PI3K/AKT pathway activation

1 Introduction Epithelial-mesenchymal transition (EMT) is defined as a complex cellular program in which epithelial cells lose polarity and intercellular adhesion while acquiring the morphology and functional traits of mesenchymal cells, manifested by enhanced migration, invasion, and resistance to apoptosis ( During the course of pulmonary fibrosis (PF), EMT is thought to exert a central regulatory role by reshaping alveolar epithelial cell phenotypes and activating fibroblasts ( Pulmonary fibrosis encompasses a spectrum of progressive chronic interstitial lung disorders, of which idiopathic pulmonary fibrosis (IPF) is the archetypal form, distinguished by repeated alveolar-epithelial insults, dysregulated EMT, and excessive fibroblast activation, leading to architectural derangement and relentless functional decline