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Graphical Abstract Similar content being viewed by others Background Ferroptosis, a regulated type of cell death resulting from iron-dependent lipid peroxidation, has been recognized as a crucial mechanism associated with the development of various diseases, including neurodegenerative, cardiovascular, digestive, musculoskeletal, and cancer-related diseases [1]
The cells were subsequently exposed to different concentrations of TVEAE for a duration of 24 h
This was an unanticipated finding since TMAO is associated with cardiovascular and metabolic disease and AD and prior human studies showed modulation of plasma TMAO by the E4 allele (Fennema et al., 2016
Treatment with NR or the PARP inhibitor Olaparib also improved mitochondrial morphology in aged nematodes and mammalian muscle cells during proteotoxic stress 114
