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Yet another reason to investigate the role of early childhood exposure to acetaminophen in the pathogenesis of autism is that eliminating acetaminophen exposure early in life is unlikely to cause net harm, regardless of the effect on autism

The fragments were incubated for 24 h under standard conditions

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Intracellular antioxidant defense involves cytosolic Cu,Zn-SOD ( SOD1 ) and the mitochondrial matrix Mn-SOD ( SOD2 ), which dismutases superoxide radical anion (O 2 ) into hydrogen peroxide (H 2 O 2 ) and oxygen (O 2 )

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