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Role of dietary fructose and hepatic de novo lipogenesis in fatty liver disease

IDO, indoleamine 2,3dioxygenase

These Ctr1-deficient mice exhibited marked growth retardation and reduced survival rates early after birth, likely due to insufficient copper absorption and subsequent impairment of copper-dependent enzymes and proteins (18)

It can be dynamically regulated, enabling an adjustment in ATP synthesis rate to meet ATP demand [10]
