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Succinate dehydrogenase mutation underlies global epigenomic divergence in gastrointestinal stromal tumor
The mechanism of actions is depicted in Figure 8
Borel MJ, Beard JL, Farrell PA
In the current study, we identified three DE clusters with distinct phenotypes that can potentially be used to identify the course and treatment targets of DE (dopamine and glutamate neurotransmitter system targeted drugs for K1 and K2, and glutamatergic drugs for K2, and antioxidants, especially for K1 and K2)
