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Nonetheless, the CNS-specific mechanisms of NAC are still only partially explored and warrant further targeted research
BCL2A1: a novel target in refractory acute myeloid leukemia with FLT3-ITD/D835 dual mutations
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In vivo studies further indicate that knockdown of AMH can accelerate follicle growth, while ectopic expression of FOXL2 can alleviate this effect, suggesting that FOXL2 and AMH exert a synergistic effect during follicle formation 93
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