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References Kane, J

New York: 1995

Heat shock protein -1 can reduce iron uptake and control capacity of the iron pool by inhibiting TFR-1 expression levels (82)

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This is possible due to the extraordinary ability of the gonococcus to modulate the interaction with different types of cervical epithelial cells in the female tract, using distinct mechanisms, including the modulation and control of distinct epithelial cell-cell adhesion complexes through the manipulation of host cell signaling and the ability to survive intracellularly within a gonococcal-containing vacuole (Edwards and Butler, 2011