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[DOI] [PMC free article] [PubMed] [Google Scholar] Colucci D.F., Buyske D.A

Collectively, our findings suggest that inflammatory eosinophils, which express high levels of Slc2a3 , act as a metabolic sink in the infected skin and thereby restrict the availability of glucose for disease-protective, highly glycolytic Th1 cells

Reduced metabolic capacity in fast and slow skeletal muscle via oxidative stress and the energy-sensing of AMPK/SIRT1 in malnutrition

There are, however, still elements that require further optimization, such as research for the new safe-harbor loci that sustain transgene expression

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