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However, the highest expression of NAT8 is in the endoplasmic reticulum of renal proximal tubular cells [179]
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When glutathione levels are depleted - as they consistently are in Hashimoto's thyroiditis, Graves' disease, and hypothyroidism - the consequences cascade: oxidative thyroid damage, protein modification, autoantigen formation, immune activation, and progressive thyroid destruction
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Glutathione regulates telomerase activity in 3T3 fibroblasts
