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10.1093/jnen/nly024 151 SensiS
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Pathological conditions such as malnutrition, critical illness, sepsis, high doses of exogenous glucocorticoids and inflammation are associated with lower IGF-I mRNA in muscle[2]
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NSBSO rapidly responded in high GSH B16 cells and consumed part of GSH, and then the resultant Nap- D F D FY-thiol and NSBSO co-assembled into nanofibers in the cell, allowing more BSO to accumulate, thereby inhibiting intracellular GSH synthesis and ultimately minimizing intracellular GSH
