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Furthermore, microRNA sequence analysis showed that IMSC-SEVs were highly enriched in miR-105-5p, which had a pivotal role in the IMSC-SEV-mediated therapeutic effect by downregulating the level of cAMP-specific hydrolase PDE4D and leading to SIRT6 activation [95] (Table 7)
Witschi A, Reddy S, Stofer B, Lauterburg BH (1992)
The initial report was of a homozygous missense mutation in exon 15, encoding the critical SH2 domain leading to aberrant folding and aggregation of the protein
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