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Subsequent research uncovered that the HGF-MET signaling pathway triggers the PI3K pathway, which in turn triggers Arg-1 expression in a dependent manner, propelling M1 macrophages to adopt an M2-like phenotype [66]

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A reduction in more than 70% in the active intestinal Ca 2+ absorption, 55% in CB D 9k expression and 90% in TRPV6 expression was observed in VDR null mice[19]

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