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During hypoxia, oxygen sensing and signal transduction are largely governed by prolyl hydroxylase domain proteins (PHD1/2/3), which hydroxylate target proteins in an oxygen-dependent manner 8,9

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Our notable exception is the observation in several studies that NAC is highly effective in elevating glutathione (GSH) under conditions where there has been a dramatic (acute depletion) drop in intracellular glutathione (GSH) levels, for instance as is the case in acetaminophen overdose