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This AP dysregulated glucose and lipid metabolism, caused mild glucose intolerance, increased body weight, and caused hepatic steatosis in the mouse model after only 4 weeks
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Keller and colleagues demonstrated a role of l-carnitine in the transcriptional regulation of genes involved in the ubiquitin proteasome system of the skeletal muscle of piglets, indicating a potential mechanism by which l-carnitine prevents muscle protein degradation [91]
