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Patients on prolonged INH therapy often receive supplemental pyridoxine to prevent peripheral neuropathy, an adverse effect related to B deficiency

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The primary mechanism of AOD-9604 in laboratory research models is selective beta-3 adrenergic receptor (beta-3 AR) activation

It was found that both AOD9604 and hGH resulted in fat loss in association with increased fat oxidation as well as increased levels of glycerol (relating to lipolysis), However, an added benefit of AOD9604 was that it did not lead to hyperglycemia in the mice or reduced insulin sensitivity

But, the quantity of cyanide is considered to be clinically irrelevant