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PubMed Beck, D.E., Sweeney, W.B., McCarter, M.D., & ipamorelin 201 Study Group
Venetoclax resistance has become increasingly evident as research progresses, primarily due to dysfunction in pro-apoptotic mechanisms and the upregulation of compensatory anti-apoptotic proteins, such as myeloid cell leukemia-1 (MCL-1)
Br J Cancer 119(7):873884
In addition, excess acetyl-CoA is transported to the cytosol either as citrate (as diagrammed in the Synthesis of Fatty Acids page) or as acetylcarnitine
