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These effects are primarily mediated through direct cellcell contact with immune cells and the secretion of paracrine factors, including cytokines, chemokines, growth factors, and other modulatory agents
MYG1 drives glycolysis and colorectal cancer development through nuclear-mitochondrial collaboration
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Moreover, a continuous release of GSH from hepatocytes to extracellular/vascular space detoxifies ROS generated by Kupffer cells, protecting the hepatic vasculature, as demonstrated in a in vivo model of partial no-flow ischemia/reperfusion injury [219]
Others have shown that VIP possesses potent anti-inflammatory effects and inhibits eosinophil migration [121]
