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Antimicrobial resistance (AMR)

This paradigm positions mtDNA heteroplasmy not merely as a passive biomarker of dysfunction, but as a programmable regulator of tumor behavior, with broad implications for targeting the LARS2 mitochondrial translationS100A8/A9 axis in cancer therapy (Fig

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Gut lymph and lymphatics: a source of factors leading to organ injury and dysfunction

This has important therapeutic implications as dedifferentiation contributes to intrinsic and acquired resistance to MAPK pathway inhibitors, and occurs as a response to inflammatory signaling during immunotherapy