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Description
Epidemiological studies have determined that Lp(a) is an independent cardiovascular risk factor, and because Lp(a) levels are determined by gene structure, Mendelian randomization studies have shown that Lp(a) has a role in causing both atherosclerosis and calcific aortic valve disease 12,13,14

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Vuksic T, Zoricic I, Brcic L, Sever M, Klicek R, Radic B, et al

G., Starodubtseva, M

, negative regulation
