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Description
P = 0.11
Secondary carnitine deficiencies are most commonly the result of trapping of carnitine in fatty acylcarnitines due to defects in mitochondrial fatty acid -oxidation enzymes
Over 90% of filtered carnitine are reabsorbed presumably in the proximal tubule by this active transporter (Figure 1)
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By blocking adenosine A1 receptors, caffeine causes dilation of the feeding arterioles in the glomeruli, thereby increasing filtration pressure
