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Dickinson DA, Forman HJ

Intriguingly, these enzymes may only scratch the surface of ER oxidative capacity, with viability and even a vestige of proinsulin biosynthesis (requisite of disulphide bond formation in the ER of endocrine pancreatic -cells) retained in dual knockouts of both ERO1 isoforms [77]

[DOI] [PMC free article] [PubMed] [Google Scholar] 75.Kozer E., Koren G

The cysteine residue in actin functions as a sensor for oxidative stress, resulting in a high level of sensitivity to ROS, RNS, and lipid peroxidation (Farah and Amberg, 2007)