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Description
Biochim Biophys Acta, 2013
always use separate syringes
High doses of the anti-rheumatic drug auranofin have been shown to activate ferroptosis by inhibiting TXNRD1 activity [55]
This Glu uptake mechanism protects against neurotoxicity caused by excessive synaptic Glu accumulation and postsynaptic overactivation (172)
CONCLUSIONS Together, this evidence suggests that oxidative stress and in particular disturbed glutathione metabolism are related to the pathogenesis of hypertension
