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Thereby, even if lipid peroxidation per se is insufficient to cause cell death, it may be considered as evidence for oxidative stress that contribute to liver injury
2) and functions of Wnt/-catenin signalling (Table 1), followed by other molecules and pathways affecting the Wnt/-catenin pathway (Table 2)
doi: 10.1111/1346-8138.12321 21
Mitochondrial damage-associated molecular patterns (DAMPS) reportedly lead to the activation of Cgas/Sting/IFN axis in renal fibrosis [92, 93]
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