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The overt phenotype of the mutant mice and blood counts were not significantly different from wildtype (Supplementary Fig

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Extracts inhibited antigen-activated degranulation in RBL-2H3 cells with an IC 50 value of 28.5 g/mL

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Specifically, oxidative stress induced by HBV activates PRKAA (the catalytic subunit of AMPK), enhancing autolysosome-dependent degradation by increasing cellular ATP levels, which depletes autophagic vacuoles [191]

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