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Other side effects reported ranged from lipohypertrophy at injection site, papilloedema (related to cranial hypertension), and facial nerve paralysis [276, 285], however symptoms did not prevail after treatment pause and restarting with lower doses [268]
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suggested that the exposure of N-retinylidene-N-retinylethanolamine (A2E), a primary element of RPE lipofuscin, to blue light fostered ferroptosis by elevating Fe 2+ levels and hindering the SLC7A11/GSH/GPX4 axis (87)
Ferroptosis is an iron-dependent mechanism of controlled cell death marked by the formation of lipid hydroperoxides [33,34]
