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All these differences on the whole may be, at least in part, responsible for the very different results observed in all the clinical and preclinical studies (33, 35)

doi: 10.1053/j.gastro.2006.08.016 97

Thus, it is possible that oxidation of Cys106 may act as the signal for DJ-1 to prevent mitochondrial dysfunction during ROS production in PD

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