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Moreover, it inhibited tunicamycin-induced cell death and protected dopaminergic neurons from the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), which triggers both oxidative and ER stress
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Macpherson, C.N.L
91, 92 What is more, the suppression of CPTI by etomoxir eventually reactivated the original low level glycolysis in prostate cancer
