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3.4 Connections between genetic mutations and metabolic changes The HPGL/PCC syndrome, where loss-of-function mutations affect SDH and increase intracellular levels of the oncometabolite succinate, thus causing onset of pheochromocytoma and paraganglioma, is a proof-of-concept that metabolic changes can drive tumorigenesis

Research Aligned Designs with Existing User Mental Models Instructors are used to thinking in terms of the classes they teach, not the way a product is built

Chem Pharmaceut Bull 24, 23642368

The intracellular and extracellular levels of S1P are spatially altered, allowing it to function as an autocrine or paracrine factor respectively, which is widely distributed in lymph and blood circulation and is believed to exist in the attenuation gradient from blood to tissues

This neuroplasticity amplifies pain signals and involves complex interactions between ascending pain signal transduction pathways, descending modulatory systems, and psychological factors [54, 55, 56, 57, 58]