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L-carnitine
PloS One 5 , e9866 (2010)
Inhibition of the gut microbial CutC/D, the TMA-generating enzyme reduced the plasma TMAO levels and rescued the platelet hyper responsiveness and thrombus formation in animal models
PATHOGENESIS Under normal conditions, MCADD patients compensate for a lack of energy production from oxidation by expending glycogen stores elsewhere in the body
Laboratory measurements Plasma glucose, insulin, and urine nitrogen Clinical biochemistry measurements were performed using standard methods on fasting samples
