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This evidence demonstrates that various clinical and pathological symptoms of AD, including the deposition of A plaques, the formation of neurofibrillary tangles, metabolic dysfunction, and cognitive decline, arise following a prolonged period of gradual accumulation of oxidative damage (Rao et al., 2020) This section will further discuss the two hypotheses on the association between A and oxidative stress

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H 2 O 2 -treated zygotes had elevated levels of ROS, as well as increased oxidative damage, activation of the G2/M checkpoint, decreased cleavage and blastocyst rates, and increased rates of apoptosis