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Excess ceramides and fatty acids disrupt the ETC, uncouple oxidative phosphorylation, and induce mitochondrial permeability transition (mPT)

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By reassessing the effects of GSH within the context of MASLDs metabolic underpinnings, investigators can generate findings that are more applicable to contemporary diagnostic and therapeutic frameworks

It should be noted that the fold changes of these redox defects are much more pronounced than that of glutamate level decrease in SLC7A11 high cancer cells under glucose starvation, and thereby correlate better with the rapid and dramatic cell death observed in these cells upon glucose starvation (Liu et al., 2020)

[DOI] [PubMed] [Google Scholar] 30.Haematocrit and the risk of developing end-stage renal disease