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Additionally, it would be valuable to investigate whether restoring iron homeostasis in CD11c + myeloid cellssuch as through iron chelator administration or inhibition of key components of iron metabolism, including IRP2can resolve inflammation and promote tissue repair
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Finally, the third burst of ERK1/2 activation associated with G2/M progression 34 was also greatly diminished and associated with a reduced number of cells replicating DNA and progressing in G2 and M phases
