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Description
The present study was also supported by funds from the AMED-CREST from the Japan Agency for Medical Research and Development, AMED and research funds from the Yamagata prefectural government and the City of Tsuruoka
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Setty, O
This polymorphism is similar to the wild type NQO1*1, results in substitutional messenger RNA splice sites that can result in a deletion of exon 4 and produce a protein deficient in quinone binding site for which enzyme activity differs according to the substrate [ 61 , 62 , 63 ]
