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Description
This understanding has resulted in substantive exploratory work on new drug targets, as well as on the concept of regulation of central carbon metabolism in cancer cells [120, 122,123,124,125]
For bone biology studies requiring absence of cortisol confounds, verify cortisol non-elevation at the experimental dose in the species and model used before attributing skeletal outcomes to GH-specific mechanisms
E1a promotes c-Myc-dependent replicative stress: implications in glioblastoma radiosensitization
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