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The cytosolic N -acetyltransferases (NATs) are responsible for the metabolism of drugs and environmental agents that contain an aromatic amine or hydrazine group
After treatment, 10% CCK-8 solution was added to each plate
METH stimulates the release of excess glutamate from neurons while inhibiting its uptake by astrocytes, leading to the overactivation of NMDARs ( tThe excitotoxicity induced by METH also triggers neuroinflammation and amplifies its neurotoxic effects
Interplay between microbiota, toll-like receptors and cytokines for the maintenance of epithelial barrier integrity
Andres E, Federici L, Affenberger S, Vidal-Alaball J, Loukili NH, Zimmer J, et al: B12 deficiency: a look beyond pernicious anemia
