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Turner DM (1994)
5.4 Wound healing BSPs exhibit significant wound-healing bioactivity, mediated primarily through activation of the TGF-/Smad signaling pathway (He X
Meyers, H
Spinal astrocytic activation contributes to mechanical allodynia in a mouse model of type 2 diabetes
The overexpression of this enzyme in the astrocytes of glial fibrillary acidic protein-heme oxygenase-1 (GFAP-HMOX1) transgenic mice resulted in increased subcortical oxidative imbalance with the induction of mitochondrial damage and autophagy, augmented dopamine and serotonin levels in the basal ganglia, and reduced dopamine 1 receptor (D1) in the nucleus accumbens, and enzyme overexpression caused degeneration of axons in the hippocampus and hyperkinetic behavior [44]
