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In addition, triptolidenol ( 25 ) was reported as well, and the result of molecular docking demonstrated its mechanism of action towards IKK [144]

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HDL-induced NO underlies many of HDLs beneficial properties to endothelial cells, including HDL-induced vasodilation, tightening of cell-to-cell junctions and increased barrier function, differentiation of endothelial progenitor cells, cell survival and proliferation, cell migration, inhibition of apoptosis, and suppression of adhesion molecule expression